How does one cure "Lockjaw"...?!


Question: How does one cure "Lockjaw"....?
is this a long process.? Any information would be appreciated!Health Question & Answer


Answers:
Some times it could be a long process done by drugs or do it yourself.
I had lockjaw and got it when having a root canal done on molars, the dentist locked it by having my mouth so wide open for a period time that when closed it locked and I caught an infection on top of it.I went to the emergency room and was treated by a doctor and back to the Dentist for counsel.I did exercises by placing medical tongue depressors, they look like popsicle sticks in my mouth everyday and wound increase the amount day by day to ease the muscle back open slowly, you increase the thickness of them until i was able to open my mouth, there are pain medications for help in doing this. This was done back in the 90's and things have change .Health Question & Answer

[edit] Neuromuscular-blocking drugs

Detailed view of a neuromuscular junction:
1. Presynaptic terminal
2. Sarcolemma
3. Synaptic vesicle
4. Nicotinic acetylcholine receptor
5. MitochondrionMain article: Neuromuscular-blocking drugs

Muscle relaxation and paralysis can theoretically occur by interrupting function at several sites, including the central nervous system, myelinated somatic nerves, unmyelinated motor nerve terminals, nicotinic acetylcholine receptors, the motor end plate, and the muscle membrane or contractile apparatus. Most neuromuscular blockers function by blocking transmission at the end plate of the neuromuscular junction. Normally, a nerve impulse arrives at the motor nerve terminal, initiating an influx of calcium ions which causes the exocytosis of synaptic vesicles containing acetylcholine. Acetylcholine then diffuses across the synaptic cleft. It may be hydrolysed by Acetylcholine esterase (AchE) or bind to the nicotinic receptors located on the motor end plate. The binding of two acetylcholine molecules results in a conformational change in the receptor that opens the sodium-potassium channel of the nicotinic receptor. This allows Na+ and Ca2+ ions to enter the cell and K+ ions to leave the cell causing a depolarization of the end plate, resulting in muscle contraction.[7] Following depolarization, the acetylcholine molecules are then removed from the end plate region and enzymatically hydrolysed by acetylcholinesterase.[5]

Normal end plate function can be blocked by two mechanisms. Nondepolarizing agents like tubocurarine block the agonist, acetylcholine, from binding nicotinic receptors and activating them, thereby preventing depolarization. Alternatively, depolarizing agents such as succinylcholine are nicotinic receptor agonists which mimic Ach, block muscle contraction by depolarizing to such an extent that it desensitizes the receptor and it can no longer initiate an action potential and cause muscle contraction.[5]These neuromuscular blocking drugs are structurally similar to acetylcholine, the endogenous ligand, in many cases containing two acetylcholine molecules linked end-to-end by a rigid carbon ring system, as in pancuronium.[5].Health Question & Answer

"Lockjaw" is a colloquial name for Tetanus. It involves the painful contraction of muscle fibers due to the bacterial toxin.
Tetanus starts with an infected wound that is not treated properly. The bacterium responsible is Clostridium tetani. It is anaerobic (i.e. air is bad for it) so deep wounds are more likely to be infected with it, since shallow wounds are more likely to get air. Still, any infected wound containing CT can develop into tetanus, if not treated properly.
The cause of the tetanus is not the bacterium itself, but a toxin that it produces.
Mild tetanus can be treated in about 10 days.
Severe tetanus requires intensive care and can take weeks to cure; or it may result in death.
So if you get a wound infection that is unusually stubborn, it's a good idea to get it checked.
But if you have had your tetanus shot, it will protect you very well.
Scooter-- I enjoy your English and I like your rodent-hood too.Health Question & Answer

WHAT YOU SHOULD KNOW
Lockjaw is an infection that cause extreme muscle stiffness and spasms. also referred to as tetanus, it's efficiently prevented next to shots of tetanus vaccine, which are usually given in infancy. Protection eventually wear stale, so a booster is recommended every 10 years.

Causes
Lockjaw is cause by germs call Clostridium tetani, which live contained by the soil. These germs typically enter the body through a puncture wound, such as one cause by stepping on a pin. Once inside the body, the germs produce a toxin that attacks the nerves in control of the muscles, thereby cause severe spasms.

Signs/Symptoms
The first symptoms typically appear inside four to 25 days after infection. At the outset, you'll be troubled by tender muscle stiffness and spasms within the jowl and nouns (from which the disease derives its name). Gradually, the vertebrae, chest, tummy, and limb also become artificial. High hallucination, convulsions, restlessness, difficulty swallowing, disorientation, sore throat, chills, and extreme distress may eventually fix together the initial symptoms

NOW. do you really own lockjaw.?

Care
Treatment includes several drugs. Muscle relaxants are prescribed to lessen muscle contractions. Antibiotics are given to fracas the infection. Antitoxin injections in the afflicted muscles give a hand halt the progress of the disease, but will not counteract the toxin already in your system or relieve symptoms already present. Intravenous nutritional fluids may also be given. If your breathing is disrupted, an artificial respirator may be needed.

http://www.healthbaba.com/Dental/1878-de...


http://www.wellsphere.com/wellpage/lockj...

http://www.reference.com/browse/lockjawHealth Question & Answer

Tetanus is progressive and can cause death if untreated. See the below websites for information on treatment options.

http://en.wikipedia.org/wiki/Tetanus#Tre...

http://www.health.state.ny.us/diseases/c...Health Question & Answer

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