what are the main syptomes of HIV?!
Question: still HIV/AIDS have not found medicns and the couse and the syptomes of HIV/ADS?
Answers:
HIV/AIDS can but NOT ALWAYS have flu like symptoms for 3 days to two weeks upon contracting it. After that time period, there are none. However, other diseases easily caught when someone's immune system is compromised are a telltale sign. They ususally appear in advanced stages of HIV/AIDS. Caposi's sarcoma, a skin cancer, can appear in lesions on the skin. Thrush, or a yeast infection in the mouth can appear (looks like a thick white coating on the toungue). Pneumonia is common. Diarrhea for lengthy stretches is also common. Most people have no symptoms until AIDS develops fully, which is why it's important to get tested often so that HIV can be treated with drugs early.
Other Answers:
anal sex..... even if you didnt have that type of sex someone linked to the person did....
None. You feel fine until lesions appear on your face, get 'run down', night sweats, aches and pains, pnemonia.
Get tested every 3 months and practise safe sex.
Take care (I am HIV- but I'm used to be a nurse).
Human Immunodeficiency Virus (HIV), member of the Retroviridae family of viruses (commonly known as retroviruses), and classified in the subfamily lentiviruses. Human infection with HIV results in a complex clinical disease known as acquired immune deficiency syndrome (AIDS), which may take ten years or more to develop. HIV was isolated in 1983 almost simultaneously by three groups of scientists: Luc Montagnier's group at the Pasteur Institute in Paris, Robert Gallo's group at the National Cancer Institute, and a group headed by Jay Levy at the University of California, San Francisco. Initial infection with HIV may cause a brief flu-like illness, which is typically followed by a long asymptomatic period during which progressive damage to the immune system occurs, resulting eventually in the onset of clinical disease.
T-Lymphocyte Infected With HIV Human immunodeficiency virus (HIV) is the cause of acquired immune deficiency syndrome (AIDS). By infecting CD4 T-lymphocytes, a type of white blood cell, HIV weakens the immune system and leaves the infected individual open to deadly infections. The viruses gain access to a T-lymphocyte by attaching to CD4 proteins on the outer surface of the cell membrane.Phototake NYC/Institut Pasteur/CNRI
HIV is closely related to viruses that cause similar immunodeficiency diseases in a range of animal species. Its origin in humans is widely accepted to have resulted from cross-species transfer of a simian immunodeficiency virus (SIV) from the chimpanzee, Pan troglodytes troglodytes, in central Africa, probably centuries ago. Changing social mores and urbanization are believed to have provided the conditions necessary for the emergence of HIV as a pandemic during the latter decades of the 20th century. In 2000, close attention to alternative aetiologic (causative) theories, such as the suggestion that HIV spread to humans from contaminated polio vaccines developed in Africa during the 1950s, concluded that there is little evidence to support them.
There are two main types of HIV, known as HIV-1 and HIV-2. HIV-1 is responsible for the majority of infections in the West, while both HIV-1 and HIV-2 are prevalent in Africa. HIV-2 is associated with a less aggressive disease course than HIV-1. HIV-1 has been further classified into several subtypes, known as clades, whose geographical distribution varies from country to country. No preventative vaccine for HIV infection exists, although several candidate vaccines have entered human trials. It remains unclear whether a single vaccine is ever likely to be able to provide protection against all HIV clades, or whether different vaccines may be required for each clade.
II STRUCTURE
Retroviruses are classified by their unique feature: the need to convert their genomic RNA into DNA (the process of reverse transcription) using an enzyme that they carry (reverse transcriptase). The outer surface of HIV is a lipid "envelope" derived from the cell membrane of infected cells. Protruding from the surface are the viral transmembrane glycoprotein (gp41) and the envelope glycoprotein (gp120) that allow HIV to bind and fuse with a target cell. Within the envelope, the viral core protein, p17, forms the matrix of the virion particle, and the core protein, p24, forms an inner cylindrically shaped nucleoid. The nucleoid contains two strands of viral genomic RNA (the genetic material of HIV) and the associated reverse transcriptase enzyme (see Genetics).
III HOW HIV CAUSES INFECTION
Life Cycle of Human Immunodeficiency Virus The human immunodeficiency virus (HIV), the cause of acquired immune deficiency syndrome (AIDS), is genetically programmed to do one thing: highjack the reproductive machinery of a human cell, then trick it into churning out as many copies of the virus as it can before the cell dies. The current best hope for the treatment of AIDS requires that patients take a number of different drugs, each of which interferes with certain steps of the HIV infection process.© Microsoft Corporation. All Rights Reserved.
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HIV infects certain human cells by binding its envelope glycoproteins gp120 and gp41 to specific molecules on the surface of the cells. Only cells that carry the appropriate molecules are susceptible to infection by HIV. In the 1980s, scientists quickly recognized that a molecule called CD4, which is found particularly on certain T-lymphocytes (a type of white blood cell), was the primary binding site, but it was only in 1996 that other co-receptors that are also required for infection were identified. Fusion of the virus with the cell membrane permits the viral nucleoid to enter the cell.
One of the co-receptors is called chemokine receptor 5 (CCR5). Because of their inherited genetic make-up, about 14 per cent of Caucasians have unusually small numbers of these receptors on the surface of their cells and a smaller proportion do not express CCR5 on their cells at all, rendering these cells less susceptible to infection with HIV. Studies suggest that this may help to explain why some people appear to be resistant to HIV infection and remain HIV-negative despite multiple exposures to the virus, and why some HIV-positive people experience slower disease progression than others.
As HIV disease progresses, HIV variants called syncytium-inducing (SI) strains evolve within the individual's body. SI variants can use an additional co-receptor on human cells, called CXCR4. This may allow HIV to infect a wider range of cells and may help to explain why the emergence of SI variants is associated with a worse prognosis. Again, a small proportion of Caucasians (about 1 per cent) do not produce this co-receptor.
Once fusion has taken place, reverse transcription then occurs to convert the viral genomic RNA into double-stranded DNA. The viral DNA is transported to the cell nucleus and is integrated, or inserted, into the normal cellular chromosomal DNA. When the right activation signals are present, the process of making new virions begins. Using the replication machinery of the host cell, the integrated viral DNA is transcribed to make messenger RNA (mRNA) and new strands of viral genomic RNA. The viral mRNA is then translated into a protein string that is cleaved into specific viral proteins. Assembly of new virions then takes place within the cell, and the new HIV particles are released by budding from the cell surface, taking a piece of the cell membrane as their envelope.
HIV replication can directly kill CD4+ T-lymphocytes. The loss of these cells paralyses the immune system and is one mechanism by which HIV infection causes AIDS.
A number of anti-HIV drugs have been developed, each targeting a different stage in this viral life cycle. By 2001, nine reverse transcriptase inhibitors and six protease inhibitors had been developed, with more in development. The widespread use of combinations of these agents in the developed world has resulted in dramatic reductions in rates of HIV-related illness and death. Several members of a new class of drugs, which inhibit the binding or fusion of HIV to host cells, are now in clinical development.
and Vertigo Deziness lazy ness Pain in all parts of the Body and HIV patient is always sleepy and trying to sleep too much.
back bone pain,vomiting, fever these are the main symptoms
The symptoms of AIDS are really symptoms which manifest in the form of other diseases which results from the breakdown of the immune system. For example, pneumonia, coughing, sores, or wound which doesn't heal too quickly.
FIRST THERE R FREE CLINICS U C AN GO TO TO C IF U HAVE IT THIS IS A BIG 1 I HAD SEVRAL SEX ED CLASSES AIDS IS HIV BUT STRONGER LEVELS OF ( I CANT REMEMBER ) BUT U CAN GO FROM HIV TO AIDS AND BACK AGIAN THERE R MEDICATIONS THAT MAKE LIFE MORE BERABLE HIV HAS NO SIGNS U HAVE TO GET TESTED IT SHOWS UP N UR SISTOM 30 DAYS AFTER GETTING INFECTED PLEASE GET HELP US PERTECTION
CONSTANTLY BEING SICK . ANTIBIOTICS DON'T HELP . SEE A DOCTOR THEY HAVE A COCKTAIL TO TAKE AND HAS BEEN SUCCESSFUL . THERE IS NO CURE FOR AIDS , ONLY PREVENTION ,,, ABSTINENCE IS THE BEST PREVENTION ..
start hating girls for first time in life.
I must tell you that the answers up to now are not accurate.
HIV does not present itself necessarily at first. It incubates for periods of time manifesting it self many different ways. I know of one case where it took over 11 years to be noticed and it was in low white blood cell count. I know another case of a child born with it who didnt have symptoms at all if her mother hadnt been diagnosed she wouldnt have known for many years.
I have included some sites for you.
Good Luck and God Bless!
You may contact me anytime I know to much about this disease.
Source(s):
http://hivinsite.ucsf.edu/hiv?page=basics-00-02
http://www.cdc.gov/hiv/pubs/faq/faq5.htm
http://www.hivtest.org/
The first symptoms of HIV infection can resemble symptoms of common cold or flu viruses. The symptoms of early infection can also be similar to the symptoms of other sexually transmitted diseases and other infections such as "mono" or hepatitis, which are much more commonly and more easily transmitted. Stress and anxiety can also produce symptoms in some people, even though they do not have HIV.
The symptoms of people who are HIV infected vary. Some examples:
# opportunistic infections are a good sign of infection (Pneumocystic Pneumonia, Kaposi's Sarcoma, Tuberculosis, Mycobacterium Avium Complex, HIV-Related Lymphoma, Toxoplasmosis Encephalitis, Cytomegalovirus Infection, Cryptocococcosis and Cryptosporidiosis)
# chronic dry, scratchy cough, shortness of breath, tightness or pressure in the chest
# rapid weight loss
# profuse night sweats
# continuous unexplained fatigue is a usual complaint of HIV/AIDS patients
# diarrhea longer than a week (found in both early and late stages of HIV)
# swollen lymph glands (lymphatic nodes in the neck, armpits and groin)
# sores, white spots or blemishes in the mouth, gums, and on the tongue is a good indication of this fungal infection - mouth ulcers (canker sores), oral candidiasis (yeast infections in the mouth or "thrush" located in the mouth and is caused by a fungus called Candida Albicans)
# burning sensation and an altered sense of taste (caused by thrush and can be easily treated with drugs such as Clotrimazole Troches and Nystatin)
# pneumonia
# excessive bruising and bleeding
# Herpes Simplex (affects the rectal, genital, and esophageal regions of the body) is a common sexually transmitted disease and may become a chronic condition in HIV/AIDS patients
# loss of appetite
# red, pink, brown or purplish blotches on and/or under the skin
# pain or difficulty swallowing
# constant headaches
# confusion or forgetfulness
# unexplainable change in vision
# Pneumococcal Pneumonia is a lung infection found usually in HIV/AIDS patients
# chronic yeast infections (women)
# pelvic inflammatory disease (women)
# cervical abnormalities (women)
# skin conditions such as rash, hives, lump, lesion, sore, spots, or abnormal growths
# chronic mono-like illness
# receding gums
# constant fevers
don't just quote any particular symptom..ok..First consult UR doc..ok..and take care.
Source(s):
I'm a Doc buddy.
HIV is a virus. Infected person can know about it after 15 years. He gets AIDS acquired [i.e.not from heredity] immune deficiency syndrome. Aids patient cannot fight minor disease like diarrhea, cough-cold & dies in two years.However HIV can be detected by medical tests in four months of infection. Pity is that HIV infected person spreads the diease from the first day of infection.
nope, no cure, no vaccine. best way to prevent it is to avoid risky behaviors such as sex with lots of people and use condoms when you do
you cant rely on symptoms to determine if you are infected or not. you need to get tested. go to a country/public clinic, they do STD testing for free.
usually flu like sympotoms for a long period of time, ie 6 weeks.
two great sites for information are below
http://www.cdc.gov/hiv
http://www.thebody.com
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